Consortium led by RIKEN solves mechanism of onset of liver fibrosis after HCV infection

The consortium led by RIKEN’s Life Science Technology Center and involving researchers from several universities found that a HCV-coded protein is cut by NS3 protease to induce a Smad activation signal which leads to the expression of TGF-ß and collagen, leading to progressing liver fibrosis.

RIKEN news release, November 27, 2013

Consortium led by RIKEN solves mechanism of onset of liver fibrosis after HCV infection
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